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Dual inhibition of AKT‐mTOR and AR signaling by targeting HDAC3 in PTEN‐ or  SPOP‐mutated prostate cancer | EMBO Molecular Medicine
Dual inhibition of AKT‐mTOR and AR signaling by targeting HDAC3 in PTEN‐ or SPOP‐mutated prostate cancer | EMBO Molecular Medicine

Aβ-Induced Drp1 phosphorylation through Akt activation promotes excessive  mitochondrial fission leading to neuronal apoptosis - ScienceDirect
Aβ-Induced Drp1 phosphorylation through Akt activation promotes excessive mitochondrial fission leading to neuronal apoptosis - ScienceDirect

PDF) Mediagebruik, e-cultuur en mediawijsheid
PDF) Mediagebruik, e-cultuur en mediawijsheid

Retracted Article: MiR-148a agomir based targeting of c-Met and Her-3 is  able to attenuate EGFR-T790M mutation driven gefitinib and erlotinib  resistan ... - RSC Advances (RSC Publishing) DOI:10.1039/C8RA10224D
Retracted Article: MiR-148a agomir based targeting of c-Met and Her-3 is able to attenuate EGFR-T790M mutation driven gefitinib and erlotinib resistan ... - RSC Advances (RSC Publishing) DOI:10.1039/C8RA10224D

Aβ-Induced Drp1 phosphorylation through Akt activation promotes excessive  mitochondrial fission leading to neuronal apoptosis - ScienceDirect
Aβ-Induced Drp1 phosphorylation through Akt activation promotes excessive mitochondrial fission leading to neuronal apoptosis - ScienceDirect

Potential role of astrocyte angiotensin converting enzyme 2 in the neural  transmission of COVID-19 and a neuroinflammatory state
Potential role of astrocyte angiotensin converting enzyme 2 in the neural transmission of COVID-19 and a neuroinflammatory state

Model D. Dosenfrosch - Publicaciones | Facebook
Model D. Dosenfrosch - Publicaciones | Facebook

Aβ-Induced Drp1 phosphorylation through Akt activation promotes excessive  mitochondrial fission leading to neuronal apoptosis - ScienceDirect
Aβ-Induced Drp1 phosphorylation through Akt activation promotes excessive mitochondrial fission leading to neuronal apoptosis - ScienceDirect

PLK4 is upregulated in prostate cancer and its inhibition reduces  centrosome amplification and causes senescence - Singh - - The Prostate -  Wiley Online Library
PLK4 is upregulated in prostate cancer and its inhibition reduces centrosome amplification and causes senescence - Singh - - The Prostate - Wiley Online Library

Dual inhibition of AKT‐mTOR and AR signaling by targeting HDAC3 in PTEN‐ or  SPOP‐mutated prostate cancer | EMBO Molecular Medicine
Dual inhibition of AKT‐mTOR and AR signaling by targeting HDAC3 in PTEN‐ or SPOP‐mutated prostate cancer | EMBO Molecular Medicine

Aβ-Induced Drp1 phosphorylation through Akt activation promotes excessive  mitochondrial fission leading to neuronal apoptosis - ScienceDirect
Aβ-Induced Drp1 phosphorylation through Akt activation promotes excessive mitochondrial fission leading to neuronal apoptosis - ScienceDirect

Mathematical Modeling Highlights the Complex Role of AKT in TRAIL-Induced  Apoptosis of Colorectal Carcinoma Cells - ScienceDirect
Mathematical Modeling Highlights the Complex Role of AKT in TRAIL-Induced Apoptosis of Colorectal Carcinoma Cells - ScienceDirect

Cells | Free Full-Text | Elevated PDK1 Expression Drives PI3K/AKT/MTOR  Signaling Promotes Radiation-Resistant and Dedifferentiated Phenotype of  Hepatocellular Carcinoma | HTML
Cells | Free Full-Text | Elevated PDK1 Expression Drives PI3K/AKT/MTOR Signaling Promotes Radiation-Resistant and Dedifferentiated Phenotype of Hepatocellular Carcinoma | HTML

Aβ-Induced Drp1 phosphorylation through Akt activation promotes excessive  mitochondrial fission leading to neuronal apoptosis - ScienceDirect
Aβ-Induced Drp1 phosphorylation through Akt activation promotes excessive mitochondrial fission leading to neuronal apoptosis - ScienceDirect

Frontiers | Phosphoinositide 3-Kinase Signaling in the Tumor  Microenvironment: What Do We Need to Consider When Treating Chronic  Lymphocytic Leukemia With PI3K Inhibitors?
Frontiers | Phosphoinositide 3-Kinase Signaling in the Tumor Microenvironment: What Do We Need to Consider When Treating Chronic Lymphocytic Leukemia With PI3K Inhibitors?

Norcycloartocarpin targets Akt and suppresses Akt-dependent survival and  epithelial-mesenchymal transition in lung cancer cells | PLOS ONE
Norcycloartocarpin targets Akt and suppresses Akt-dependent survival and epithelial-mesenchymal transition in lung cancer cells | PLOS ONE

Aβ-Induced Drp1 phosphorylation through Akt activation promotes excessive  mitochondrial fission leading to neuronal apoptosis - ScienceDirect
Aβ-Induced Drp1 phosphorylation through Akt activation promotes excessive mitochondrial fission leading to neuronal apoptosis - ScienceDirect

Dual inhibition of AKT‐mTOR and AR signaling by targeting HDAC3 in PTEN‐ or  SPOP‐mutated prostate cancer | EMBO Molecular Medicine
Dual inhibition of AKT‐mTOR and AR signaling by targeting HDAC3 in PTEN‐ or SPOP‐mutated prostate cancer | EMBO Molecular Medicine

Cells | Free Full-Text | Elevated PDK1 Expression Drives PI3K/AKT/MTOR  Signaling Promotes Radiation-Resistant and Dedifferentiated Phenotype of  Hepatocellular Carcinoma | HTML
Cells | Free Full-Text | Elevated PDK1 Expression Drives PI3K/AKT/MTOR Signaling Promotes Radiation-Resistant and Dedifferentiated Phenotype of Hepatocellular Carcinoma | HTML

Retracted Article: MiR-148a agomir based targeting of c-Met and Her-3 is  able to attenuate EGFR-T790M mutation driven gefitinib and erlotinib  resistan ... - RSC Advances (RSC Publishing) DOI:10.1039/C8RA10224D
Retracted Article: MiR-148a agomir based targeting of c-Met and Her-3 is able to attenuate EGFR-T790M mutation driven gefitinib and erlotinib resistan ... - RSC Advances (RSC Publishing) DOI:10.1039/C8RA10224D

A functional genetic screen defines the AKT-induced senescence signaling  network | Cell Death & Differentiation
A functional genetic screen defines the AKT-induced senescence signaling network | Cell Death & Differentiation

Cancers | Free Full-Text | Acquired Mechanisms of Resistance to  Osimertinib—The Next Challenge | HTML
Cancers | Free Full-Text | Acquired Mechanisms of Resistance to Osimertinib—The Next Challenge | HTML

PLK4 is upregulated in prostate cancer and its inhibition reduces  centrosome amplification and causes senescence - Singh - - The Prostate -  Wiley Online Library
PLK4 is upregulated in prostate cancer and its inhibition reduces centrosome amplification and causes senescence - Singh - - The Prostate - Wiley Online Library

Evolutionary Inference Predicts Novel ACE2 Protein Interactions Relevant to  COVID-19 Pathologies
Evolutionary Inference Predicts Novel ACE2 Protein Interactions Relevant to COVID-19 Pathologies